QUIZ NUMBER 42 |
pH of amniotic fluid is: |
a. 6.2 |
b. 6.8 |
c. 7.2 |
d. 8.2 |
ANSWER NUMBER 42 |
c. 7.2 |
WINNERS QUIZ NUMBER 42 |
FIRST PRIZE
Dr. Kiran Dabholkar
9167008017 |
SECOND PRIZE
Dr Razia Mansuri
9820739507 • drraziamansuri@rediffmail.com
|
THIRD PRIZE
Dr Sameen Mansuri
8108087304 • sameen2001@rediffmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 41 |
Identify |
 |
ANSWER NUMBER 41 |
Answer : 3 D ultrasound showing septate uterus |
WINNERS QUIZ NUMBER 41 |
FIRST PRIZE
Dr Madhuri V Joshi
09869046424 |
SECOND PRIZE
Dr. Prachi Risbud
7738535015 |
THIRD PRIZE
Dr. Kiran Dabholkar
9167008017 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 40 |
Identify (USG of Uterus) |
 |
ANSWER NUMBER 40 |
USG of the uterus showing snowstorm appearance in case of complete vesicular mole
|
WINNERS QUIZ NUMBER 40 |
FIRST PRIZE
Dr Shilpa Agrawal
9223280008 |
SECOND PRIZE
Dr. Nandini Ram Babu
9892911137 |
THIRD PRIZE
Dr. Prachi Risbud
7738535015 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 39 |
Fetal pulmonary maturity can be evaluated by phospholipids’ activity in amniotic fluid. In which of the following pregnancies does the fetus have the least chance of developing respiratory distress syndrome (RDS)? |
a. Normal pregnancy: amniotic fluid L/S is 1.8:1, phosphatidyl glycerol (PG) is absent |
b. Diabetic pregnancy: amniotic fluid L/S is 2:1, PG is absent |
c. Diabetic pregnancy: amniotic fluid L/S is 2:1, PG is present |
d. All of the above |
ANSWER NUMBER 39 |
c. Diabetic pregnancy: amniotic fluid L/S is 2:1, PG is present |
WINNERS QUIZ NUMBER 39 |
FIRST PRIZE
Dr. Razia Mansuri
9820739507 • drraziamansuri@rediffmail.com |
SECOND PRIZE
Dr. Sameen Mansuri
8108087304 • sameen2001@rediffmail.com |
THIRD PRIZE
Dr. Nandini Ram Babu
9892911137 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 38 |
The category 4 for IUCD are all, EXCEPT: |
a. Submucous fibroid |
b. Puerperal sepsis |
c. Heart disease |
d. Acute PID |
ANSWER NUMBER 38 |
c. Heart disease |
WINNERS QUIZ NUMBER 38 |
FIRST PRIZE
Dr. Isha Jain
9425110522
|
SECOND PRIZE
Dr. Razia Mansuri
9820739507 • drraziamansuri@rediffmail.com |
THIRD PRIZE
Dr. Sameen Mansuri
8108087304 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 37 |
Which of the following is known as pregnancy tumor? |
a. Pyogenic granuloma |
b. melanoma |
c. Molar pregnancy |
d. All of the above |
ANSWER NUMBER 37 |
a. Pyogenic granuloma |
WINNERS QUIZ NUMBER 37 |
FIRST PRIZE
Dr. Razia Mansuri
9820739507 • drraziamansuri@rediffmail.com
|
SECOND PRIZE
Dr. Sameen Mansuri
8108087304 • sameen2001@rediffmail.com |
THIRD PRIZE
Dr. Kiran Dabholkar
9167008017 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 36 |
If fetus is suffering from hypoxia, which of the biophysical profile (BPP) parameter will be affected last? |
a. Fetal tone |
b. Fetal breathing movement |
c. Fetal movements |
d. NST |
ANSWER NUMBER 36 |
a. Fetal tone |
CNS centers that may regulate the biophysical activities appear to vary in their sensitivity to hypoxia. So, a “gradual
hypoxia” model has been proposed. This model states that the activities that first appear embryologically are the last to
disappear with progressively worsening hypoxia. |
For example, the first activity to appear, fetal tone at about 7.5–8.5 weeks, is also presumably the last activity to disappear
with progressively worsening hypoxia. Fetal movements develop after fetal tone (at 9 weeks) and then fetal breathing (at
20–21 weeks), and finally FHR reactivity, at approximately 28 weeks.
|
According to the gradual hypoxia model, FHR-R would be the first component of the BPP to become abnormal (i.e., to be
lost) in hypoxic states. This gradual hypoxia concept would help explain the increased incidence of abnormal fetal outcome as more biophysical activities decrease in occurrence and eventually disappear. So finally the persistent and true lack of Fetal Tone would be associated with the highest perinatal death rate. |
WINNERS QUIZ NUMBER 36 |
FIRST PRIZE
Dr. Prachi Risbud
7738535015
|
SECOND PRIZE
Dr.Isha Jain
9425110522 • drishabharatjain@gmail.com |
THIRD PRIZE
Dr. Nandini Ram Babu
9892911137 • atg2016nandini@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 35 |
Which of the following fetal complications is associated with the penicillamine use in pregnancy? |
a. Conradi syndrome |
b. Renal anomalies |
c. Thymus hypoplasia |
d. Cutis laxa |
ANSWER NUMBER 35 |
d. Cutis laxa |
Pencillamine interferes with the synthesis of collagen and elastin and can cause elastosis perforans serpiginosa and
localized cutis laxa. |
WINNERS QUIZ NUMBER 35 |
FIRST PRIZE
priyanka honavar
priyankahonavar@yahoo.co.in
|
SECOND PRIZE
Priya Shah
dr_pnshah@icloud.com |
THIRD PRIZE
Dr. Prachi Risbud
7738535015 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 34 |
Maximum permissible dose of radiation in pregnancy is: |
a. 0.05 rads |
b. 0.5 rads |
c. 5 rads |
d. 10 rads |
ANSWER NUMBER 34 |
c. 5 rads |
The harmful fetal effects of ionizing radiation have been extensively studied for cell damage with resultant dysfunction of
embryogenesis.
The risk is greatest at 8–15 weeks, and larger doses are necessary at 16–25 weeks to cause an equivalent proportion of cases
of mental retardation.
Current evidence suggests that there is no increased risk of malformations, growth restriction, or abortion from a radiation
dose of 5 rads or less. |
WINNERS QUIZ NUMBER 34 |
FIRST PRIZE
Dr Nandini Ram Babu
9892911137 • atg2016nandini@gmail.com
|
SECOND PRIZE
Dr. Prachi Risbud
7738535015 |
THIRD PRIZE
Shilpa Agrawal
drshilpaagrawal@yahoo.co.in |
Winners Please Contact MOGS Office |
QUIZ NUMBER 33 |
IDENTIFY |
 |
ANSWER NUMBER 33 |
ESSURE CONTRACEPTION |
WINNERS QUIZ NUMBER 33 |
FIRST PRIZE
Dr. Kiran Dabholkar
kmdabholkar@gmail.com
|
SECOND PRIZE
Dr. Freni Shah
frenishah9488@yahoo.in |
THIRD PRIZE
Dr. Siddesh Iyer
siddeshiyer@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 32 |
IDENTIFY |
 |
ANSWER NUMBER 32 |
ANENCEPHALY |
WINNERS QUIZ NUMBER 32 |
FIRST PRIZE
Dr. Kiran Dabholkar
kmdabholkar@gmail.com
|
SECOND PRIZE
Dr. Prachi Risbud
7738535015 |
THIRD PRIZE
Dr. Shweta More
shwetaankushmore@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 31 |
The placenta of twins can be: |
a. Dichorionic and monoamnionic in dizygotic (DZ) twins |
b. Dichorionic and monoamnionic in monozygotic (MZ) twins |
c. Monochorionic and monoamnionic in DZ twins |
d. Dichorionic and diamniotic in MZ twins |
ANSWER NUMBER 31 |
d. Dichorionic and diamniotic in MZ twins |
WINNERS QUIZ NUMBER 31 |
FIRST PRIZE
Dr. Shilpa Agrawal
9223280008
|
SECOND PRIZE
Dr. Ashwini Sakhalkar
ashwinisakhalkar@gmail.com |
THIRD PRIZE
Dr. Rashmi G. Jalvee
983391883
|
Winners Please Contact MOGS Office |
QUIZ NUMBER 30 |
"TIP OF ICEBERG" sign on USG is seen in : |
a. Adenomyosis |
b. Dermoid Cyst |
c. Ectopic pregnancy |
d. All of the above |
ANSWER NUMBER 30 |
b. Dermoid Cyst |
Tip of the iceberg sign (on USG) refers to one of the characteristic appearances of an ovarian dermoid cyst. If there are echogenic contents of sebum/hair, they cause marked posterior acoustic attenuation so that only the superficial part of the cyst is seen (like an iceberg, one may only be able to see a small piece of the structure, with a much larger piece deeper). The intense acoustic shadowing in a dermoid may also be caused by calcifications (such as teeth) or by fat in a Rokitanskynodule. |
WINNERS QUIZ NUMBER 30 |
FIRST PRIZE
Dr. Divya R
7039060390 • rdtmc06@gmail.com
|
SECOND PRIZE
Dr. Shilpa Agrawal
9223280008 |
THIRD PRIZE
Dr. Nandini Ram Babu
9892911137 • atg2016nandini@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 29 |
Trial of scar is contraindicated in all EXCEPT :
|
a. History of previous classical Cesarean Section |
b. History of previous Cesarean section due to contracted pelvis |
c. Previous 3 LSCS |
d. History of previous LSCS due to malpresentation |
ANSWER NUMBER 29 |
d. History of previous LSCS due to malpresentation |
WINNERS QUIZ NUMBER 29 |
FIRST PRIZE
Kiran Dabholkar
9167008017
|
SECOND PRIZE
Dr. Freni Shah
9869562008 |
THIRD PRIZE
Dr. Prachi Risbud
7738535015 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 28 |
A 30-year-old primigravida at 39 weeks has been completely dilated and has been pushing for 3 h. She had taken epidural analgesia. She is exhausted and has a temperature of 37.8°C. The fetal heart rate is 170/min with decreased
variability. The patient’s membranes have been ruptured for over 24 h. The patient’s cervix is completely dilated and effaced, and the fetal head is visible at the introitus between pushes. The fetal bones are at the +3 station. What is the most appropriate next step in the management of this patient?
|
a. Deliver the patient by cesarean section |
b. Encourage the patient to continue to push after a short rest |
c. Attempt operative delivery with forceps |
d. Apply fundal pressure |
ANSWER NUMBER 28 |
c. Attempt operative delivery with forceps |
Indications for an operative vaginal delivery with a vacuum extractor or forceps occur in situations where the fetal head
is engaged, the cervix is completely dilated, and there is a prolonged second stage, suspicion of potential fetal compromise,
or need to shorten the second stage for maternal benefit. In this situation, all the indications for operative delivery apply.
This patient has been pushing for 3 h, which is the definition for prolonged second stage of labor in a nulliparous patient
with an epidural. In addition, potential maternal and fetal compromise exists, since the patient has the clinical picture of
chorioamnionitis and the fetal heart rate is not reassuring. It is best to avoid cesarean section, since it would take more time to
achieve and since the patient is infected. At full dilatation and a suitable station, forceps is faster than LSCS in baby delivery.
|
WINNERS QUIZ NUMBER 28 |
FIRST PRIZE
Shilpa Agrawal
9223280008
drshilpaagrawal@yahoo.co.in |
SECOND PRIZE
Dr. Freni Shah
9869562008 |
THIRD PRIZE
Dr. Nandini Ram Babu
9892911137 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 27 |
69 yrs old patient with Ovarian Cancer, when explored shows bilateral ovarian involvement with presence of ascites with positive washings for malignant cells with retroperitoneal Lymph Nodes involvement.
As per FIGO staging this is stage : |
a. I C3 |
b. III A |
c. IVA |
d. II B |
ANSWER NUMBER 27 |
b. III A |
As per the old FIGO staging its stage IIIC, but as per latest 2014 staging, its stage IIIA
Pleural effusion with positive cytology is stage IV A |
WINNERS QUIZ NUMBER 27 |
FIRST PRIZE
Kiran Dabholkar
kmdabholkar@gmail.com |
SECOND PRIZE
Dr. Prachi Risbud
7738535015 |
THIRD PRIZE
Bhavini Shah
doc.bhavini@gmail.com
|
Winners Please Contact MOGS Office |
QUIZ NUMBER 26 |
A 28-year-old lady had Copper T inserted 2 years back. Now Cu-T threads are not seen. USG shows Cu T partly in abdominal cavity. Method of removal is: |
a. Hysteroscopy |
b. No need of removal /wait and watch |
c. IUCD hook |
d. Laparoscopy |
ANSWER NUMBER 26 |
d. Laparoscopy |
Explanation: Copper can cause inflammatory reaction and can cause intestinal obstruction. Therefore, never wait and watch. When Cu T is embedded within uterine cavity, hysteroscopic removal is the method of choice. It is preferred over IUCD hook. Hysteroscopy cannot visualize the Cu T that is in the abdominal cavity. However, when IUCD enters the abdominal cavity (partly or completely), laparoscopy is the preferred modality for retrieval.
|
WINNERS QUIZ NUMBER 26 |
FIRST PRIZE
Dr. Freni Shah
9869562008 |
SECOND PRIZE
Dr. Shilpa Agrawal
9223280008 |
THIRD PRIZE
Dr. Rashmi G. Jalvee
9833918836
|
Winners Please Contact MOGS Office |
QUIZ NUMBER 25 |
There is loss of knee jerks when magnesium sulfate concentration reaches: |
a. 5–7 mEq/L |
b. 10–11 mEq/L |
c. 8–10 mEq/L |
d. >12 mEq/L |
ANSWER NUMBER 25 |
b. 10–11 mEq/L |
Explanation: Magnesium sulfate is the treatment of choice for the prevention and treatment of eclamptic seizures. It reduces motor end plate sensitivity to acetyl choline. It induces cerebral vasodilatation, dilates uterine arteries, increases production of endothelial prostacyclin, and inhibits platelet activation. Magnesium may also prevent seizures by interacting with N-methyl-
D-aspartate (NMDA) receptors in the central nervous system. |
After the initial dose, repeat injections are given only if knee jerks are present, urine output exceeds 30 mL/h, and respiration rate is more than 12/min. |
- The therapeutic level of serum magnesium is 4–7 mEq/L.
- 8–10 mEq/L = uterus stops contracting.
- Patellar reflux disappears when magnesium level reaches above 10 mEq/L (12 mg/dL), presumably because of curariform action.
- This sign serves to warn of impending magnesium toxicity, because a further increase leads to respiratory depression.
- When magnesium levels reach >12 mEq/L, respiratory depression develops, and respiratory paralysis and arrest follow.
- Treatment is with calcium gluconate 1 g IV and withholding magnesium sulfate reverse mild-to-moderate respiratory depression.
|
WINNERS QUIZ NUMBER 25 |
FIRST PRIZE
Dr. Kiran Dabholkar
098192 17200 |
SECOND PRIZE
Dr. Freni Shah
frenishah9488@yahoo.in |
THIRD PRIZE
Dr. Prachi Risbud
7738535015
|
Winners Please Contact MOGS Office |
QUIZ NUMBER 24 |
The following on USG indicates monozygotic twin pregnancy : |
a. 2 separate placenta |
b. Twin peak sign |
c. Siamese twins |
d. Different fetal sex |
ANSWER NUMBER 24 |
c. Siamese twins |
All siamese twins are Monozygotic |
MZ twins are of following varieties depending upon the time of twinning:
1. Within 72 hours of fertilization = DC, DA
2. Between 4th & 8th day = MC, DA
3. Between 8th & 12th day = MC, MA
4. After 12 days = conjoint/Siamese twins
Twin peak sign indicated 2 fused placenta (seen in dichorionic twins) |
WINNERS QUIZ NUMBER 24 |
FIRST PRIZE
Dr. Freni Shah
frenishah9488@yahoo.in |
SECOND PRIZE
Dr. Shilpa Agrawal
drshilpaagrawal@yahoo.co.in |
THIRD PRIZE
Dr. Prachi Risbud
7738535015
|
Winners Please Contact MOGS Office |
QUIZ NUMBER 23 |
The production rate of the following hormone near term, is the greatest of any known hormone in humans (approximately 1gm/day): |
a. Relaxin |
b. Progesterone |
c. hCG |
d. hPL |
ANSWER NUMBER 23 |
d. hPL |
Explanation:
Human placenta lactogen (hPL) was named so , because of its potent lactogenic & growth hormone like bioactivity, as well as immunochemical resemblance to human growth hormone.
It is detected as early as 2nd or 3rd week after fertilization.
It is a single , nonglycosylated polypeptide chain with a molecular weight of 22,279 Da. It is derived from a 25,000 Da precursor.
Maternal plasma concentrations are linked to placental mass & they rise steadily till 34-36 weeks of gestation.
The hPL production rate near term is by far the greatest of any known hormone in humans- approximately 1gm/day.
The half-life in maternal plasma is between 10 & 30 minutes
The functions of hPL are:
- It decreases maternal insulin sensitivity (increases insulin resistance) & ensures nutrient flow to the fetus.
- It promotes maternal lipolysis with the release of free fatty acids. This provides an energy source for maternal metabolism & fetal nutrition.
It favors protein synthesis & provides a readily available amino acid source to the fetus.
- Potent angiogenic hormone, & may serve a function in fetal vasculature formation
|
WINNERS QUIZ NUMBER 23 |
FIRST PRIZE
Dr. Prachi Risbud
7738535015
|
SECOND PRIZE
Dr. Monika Nathani
9075013533
|
THIRD PRIZE
Dr. Nandini Ram Babu
9892911137
|
Winners Please Contact MOGS Office |
QUIZ NUMBER 22 |
Infants of diabetic mother are likely to have which of the following cardiac anomaly? |
a. Coarctation of aorta |
b. Fallot’s tetrology |
c. Ebstein’s anomaly |
d. Transposition of great arteries |
ANSWER NUMBER 22 |
d. Transposition of great arteries |
Explanation:
Incidence of major congenital malformation in children of diabetic mothers is 5–10%, and most common defects are neural defects (such as anencephaly, spina bifida, and encephalocele) cardiac defects (VSD, transposition of great vessels).
Caudal regression syndrome/sacral agenesis is a defect most specific to diabetic embryopathy.
Hyperglycemia probably increases the development of free oxygen radicals and interferes with arachidonic acid
metabolism, which are responsible for embryopathy.
Major Birth Defects in Infants of Diabetic Mothers :
- Neural tube defects(anencephaly, spina bifida)
- VSD, ASD
- Renal agenesis
- Duodenal atresia
- Single umbilical artery
- Microcephaly
- Transposition of great vessels
- Hydronephrosis
- Anorectal atresia
- Sacral agenesis
- HOCM
- Ureteral duplication
|
WINNERS QUIZ NUMBER 22 |
FIRST PRIZE
Dr. Shilpa Agarwal
9223280008
|
SECOND PRIZE
Dr. Kiran Dabholkar
9167008017
|
THIRD PRIZE
Dr. Nirmal khare
9818427823
|
Winners Please Contact MOGS Office |
QUIZ NUMBER 21 |
16-year-old girl presents with 6 × 6 cm right ovarian mass with absent AFP, negative CA125, and increased alkaline
phosphatase. Diagnosis is: |
a. Dysgerminoma |
b. Mucinous cystadenocarcinoma |
c. Endodermal sinus tumor |
d. Teratoma |
ANSWER NUMBER 21 |
a. Dysgerminoma |
Dysgerminoma is the MC malignant germ-cell tumor. Once diagnosed, dysgerminomas respond well to therapy,
potentially sparing patients from infertility and early mortality.
The exact etiology of dysgerminomas has not been determined, although recent molecular studies have implicated loss of
function with potential tumor suppressor gene TRC8/RNF139 as a possible etiology.
Germ-cell tumors generally occur in the first 2 decades of life.
Epithelial tumors occur in perimenopausal and postmenopausal ladies. |
Ovarian Tumor |
Endodermal sinus /yolk sac |
Epithelial |
Dysgerminoma |
Choriocarcinoma |
Granulosa cell |
WINNERS QUIZ NUMBER 21 |
FIRST PRIZE
Dr. Nisha Krishnan
9076547783
nisha.obgy@gmail.com |
SECOND PRIZE
Dr. Shilpa Kaushal
7506012801
shilpakaushal55@gmail.com |
THIRD PRIZE
Dr. Monika Nathani
monika.nathani@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 20 |
36 weeks pregnant lady complains of bleeding PV since 4 hours. Hb 6gm%, BP-90/60, P/A-uterus tonically contracted, FHS absent. BT=7min & CT= 14min. The likely diagnosis as per Page’s classification is : |
a. Grade I Abruptio |
b. Grade II Abruptio |
c. Grade III Abruptio |
d. Grade IV Abruptio |
ANSWER NUMBER 20 |
ANSWER : GRADE 3 ABRUPTION |
Page’s Classification for Abruptio Placentae
Grade 0 :Retrospective diagnosis (after delivery)
Grade 1 : External bleeding, uterine tenderness, and no fetal distress
Grade 2 : Fetal distress or IUFD
Grade 3 : Maternal shock, with or without DIC
|
WINNERS QUIZ NUMBER 20 |
FIRST PRIZE
Dr. Freni Shah
frenishah9488@yahoo.in |
SECOND PRIZE
Dr. Rakhi Sikarwar
rakhisikarwar4@gmail.com |
THIRD PRIZE
Dr. Asmita More
8767460154
asmitamore324@gmail.com |
FOURTH PRIZE
Dr. Saumya Niviti
saumyaniviti03@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 19 |
TRUE about CARBETOCIN is : |
a. It can be administered intravenously or intramuscularly. |
b. The recommended dose for an average adult female is 1000 micrograms |
c. Carbetocin can be used to induce or augment labor |
d. All of the above |
ANSWER NUMBER 19 |
a. It can be administered intravenously or intramuscularly. |
Carbetocin is a newer drug used to control PPH particularly following Cesarean section. It is an eight amino acid long analogue of oxytocin (a nonapeptide) and thus has a similar action. It can be administered intravenously or intramuscularly. The recommended dose for an average adult female is 100 ug, administered slowly over a minute. Contractile effects of the uterus are apparent within two minutes and can be observed for approximately one hour, though maximum binding occurs about 30 minutes after intramuscular injection. Administration can be performed only once; further administration would prove risky. If further uterine stimulation is needed, treatment with other forms of oxytocic uterotonic drugs should be used
Endogenous and synthetic oxytocin has a half-life of approximately 3.5 minutes. Carbetocin, in comparison, has a much longer half-life ranging from 85–100 minutes. The elimination half-life following intravenous administration is around 40 minutes Carbetocin should not be used to induce or augment labor since it could cause cardiac or respiratory distress to mother or infant |
WINNERS QUIZ NUMBER 19 |
FIRST PRIZE
Dr. Saumya Niviti
9967769717
saumyaniviti03@gmail.com |
SECOND PRIZE
Dr. Rukhsana Mhate
9820645014 |
THIRD PRIZE
Dr. Ashwini Ingale
8087593134
dr.ashwini_ingle@rediffmail.com |
FOURTH PRIZE
Dr. Sumati Saxena
mailmesumatisaxena@rediffmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 18 |
Identify the abnormality |
 |
ANSWER NUMBER 18 |
Battledore placenta is a placenta where there is marginal insertion of the umbilical cord called because of the fancied resemblance to the racquet used in badminton |
WINNERS QUIZ NUMBER 18 |
FIRST PRIZE
Dr. Sonam Kataria
9967769717
sonam_kataria777@yahoo.com |
SECOND PRIZE
Dr. Kiran Dabholkar
kmdabholkar@gmail.com |
THIRD PRIZE
Dr. Kranti Phadnis
krantiphadnis@gmail.com |
FOURTH PRIZE
Dr. Sumati Saxena
9452495704
saumyaniviti03@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 17 |
Identify this image |
 |
ANSWER NUMBER 17 |
Cu-Fix IUD (Flexi-Gard): This is frameless IUD consisting of six copper sleeves (300 mm2 of copper) strung on a surgical polypropylene nylon thread, which is knotted at the upper end. |
WINNERS QUIZ NUMBER 17 |
FIRST PRIZE
Dr. Bhavini Shah
doc.bhavini@gmail.com |
SECOND PRIZE
Dr. Parikshit Tank
pariktank@gmail.com |
THIRD PRIZE
Dr. Deepa Talreja
7506987141 |
FOURTH PRIZE
Dr. Jatinder Kaur
9892590464 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 16 |
Identify the anomaly in this USG image of fetal abdomen |
 |
ANSWER NUMBER 16 |
Explanation:
The double bubble sign is seen in infants and represents dilatation of the proximal duodenum and stomach. It is seen in both radiographs and ultrasound, and can be identified antenatally.
Causes include :
- Congenital obstruction
- duodenal web
- duodenal atresia
- duodenal stenosis
- annular pancreas
- midgut volvulus
- external compression of the dudenum
|
WINNERS QUIZ NUMBER 16 |
FIRST PRIZE
Dr. Shilpa Agrawal
+91 92232 80008
drshilpaagrawal@yahoo.co.in |
SECOND PRIZE
Dr. Bhavini Shah
doc.bhavini@gmail.com |
THIRD PRIZE
Dr. Dhaval Belvi 9820273729 |
FOURTH PRIZE
Dr. Sachin Dalal
9833032120 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 15 |
A pregnant lady presents with jaundice and distension and pedal edema after delivering normal baby. Her clinical condition deteriorates with increasing abdominal distension and severe ascites. Her bilirubin is 5 mg/dL, S. alkaline phosphatase was 450 u/L and ALT 345 IU/L. There is tender hepatomegaly 6 cm below costal margin and ascitic fluid shows protein 3 mg%. Diagnosis is: |
a. Preeclampsia |
b. Acute fatty liver of pregnancy |
c. HELLP syndrome |
d. Budd-Chiari syndrome |
ANSWER NUMBER 15 |
d. Budd-Chiari syndrome |
Explanation:
Budd-Chiari syndrome, a disorder characterized by thrombotic occlusion of the hepatic veins, is a rare complication of pregnancy. Most reported cases presented within a few weeks of delivery, but in several cases onset occurred during pregnancy. The increased synthesis of factors II, VII, and X, as well as of fibrinogen observed in normal pregnancy, may be a predisposing factor. The onset may be acute, with the rapid development of abdominal pain and distension and sometimes jaundice. There is tender hepatomegaly, and ascites of high protein content is almost always present. Aminotransferases are often markedly raised when the onset is rapid, but jaundice is present in only half the cases.Treatment is often unsatisfactory, and the prognosis guarded.
:
|
WINNERS QUIZ NUMBER 15 |
FIRST PRIZE
Dr. Dhaval Belvi
9833700719 |
SECOND PRIZE
Dr. Kiran Dabholkar
9167008017 |
THIRD PRIZE
Dr. Rajneet Bhatia rajneet1910@gmail.com |
FOURTH PRIZE
Dr. Ranjit Inamdar
9820273729 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 14 |
A female presents with leaking and meconium stained liquor at 32 weeks of gestation. Which of the following organism would be responsible: |
a. CMV |
b. Listeria |
c. Toxoplasma |
d. Rubella |
ANSWER NUMBER 14 |
b. Listeria |
Explanation:
Listerial infections are more common in very old or young, pregnant or immunocompromised patients. Listeriosis during pregnancy can have the following effects:
|
Maternal
- Fever
- Pyelonephritis
- Meningitis
- Preterm labor
Fetal
- Discolored, brownish or Meconium Stained Amniotic Fluid even with preterm gestations. (Generally MSAF is seen with postdatism and is very rarely seen with preterm labor)
- Chorioamnionitis
- Fetal infection: disseminated granulomatous lesions with microabscesses
- IUFD
- Neonatal sepsis and mortality
CMV, Rubella and Toxoplasma infections have a teratogenic effect and are unlikely to cause preterm labor and MSAF.
|
WINNERS QUIZ NUMBER 14 |
FIRST PRIZE
Dr. Pooja Kale
9029080799 kpoo87@gmail.com |
SECOND PRIZE
Dr. Bhavini Shah doc.bhavini@gmail.com
|
THIRD PRIZE
Dr. Shilpa Agrawal
drshilpaagrawal@yahoo.co.in |
FOURTH PRIZE
Dr. Freni Shah
frenishah9488@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 13 |
An intra-uterine pregnancy of approximately 10 weeks’ gestation is confirmed in a 30-year-old with an IUCD in place. The patient expresses a strong desire for the pregnancy to be continued. On examination, the string of the IUCD is noted to be protruding from the cervical os. The most appropriate course of action is to: |
a. Leave the IUCD in place without any other treatment |
b. Remove the IUCD to decrease the risk of malformations |
c. Remove the IUCD to decrease the risk of infection |
d. Terminate the pregnancy because of the high risk of malformations |
ANSWER NUMBER 13 |
c. Remove the IUCD to decrease the risk of infection |
Explanation: Although there is an increased risk of spontaneous abortion, and a small risk of infection, an intra-uterine pregnancy can occur and continue successfully to term with an IUD in place. However, if the patient wishes to keep the pregnancy and if the string is visible, the IUD should be removed in an attempt to reduce the risk of infection, abortion, or both. An IUD in situ does not cause any malformations/anomalies in the fetus. WHO recommends that if the IUD can be removed easily it should be removed to reduce the risk of infection and abortion.
If the IUD cannot be removed easily, it can be kept in situ and it will be expelled after placental delivery. |
WINNERS QUIZ NUMBER 13 |
FIRST PRIZE
Kranti Phadnis
krantiphadnis@gmail.com |
SECOND PRIZE
Dhaval Belvi
9833700719 |
THIRD PRIZE
Bhavini Shah
doc.bhavini@gmail.com |
FOURTH PRIZE
Shilpa Agrawal
drshilpaagrawal@yahoo.co.in |
Winners Please Contact MOGS Office |
QUIZ NUMBER 12 |
Identify the fetal anomaly |
 |
ANSWER NUMBER 12 |
Cystic hygroma (CH) refers to a type of congenital lymphangioma |
Epidemiology: They usually occur in the fetal/infantile and paediatric populations with most lesions presenting by the age of two. The estimated prevalence in the fetal population is 0.2-3%. |
Radiographic features: They are usually well circumscribed and are of fluid density. Cystic hygromas may also have an infiltrative appearance, and may be uni or multilocular. The density can also be variable with a combination of fluid, soft-tissue density and fat. |
Antenatal Ultrasound: On antenatal ultrasound they may present as a nuchal cyst and may show septations +/- evidence of fetal anasarca/hydrops fetalis. The presence of septations may indicate a poorer outcome |
WINNERS QUIZ NUMBER 12 |
FIRST PRIZE Dr. Freni Shah
frenishah9488@gmail.com |
SECOND PRIZE
Dr. Shilpa Kaushal
7506012801 |
THIRD PRIZE
Dr. Shilpa
9223280008 • drshilpaagrawal@yahoo.co.in |
FOURTH PRIZE
Dr. Jatinder Kaur
drjatinder75@yahoo.co.in |
Winners Please Contact MOGS Office |
QUIZ NUMBER 11 |
Identify this Stalwart |
 |
ANSWER NUMBER 11 |
James Marion Sims (January 25, 1813 – November 13, 1883) was an American physician and a pioneer in the field of surgery, known as the "father of modern gynecology". His most significant work was to develop a surgical technique for the repair of vesicovaginal fistula |
Contributions: |
- Vaginal surgery: fistula repair.
- Instrumentation: Sims' speculum; Sims' sigmoid catheter.
- Exam and surgical positioning: Sims' position.
- Fertility treatment: Insemination and postcoital test.
- Cancer care: Sims argued for the admission of cancer patients to the Woman's Hospital, at a time when people opposed him, as they feared that cancer was contagious.
- Abdominal surgery: Sims advocated that in the case of an abdominal bullet wound, a laparotomy was needed to stop bleeding, repair the damage and drain the wound. His opinion was sought when President James Garfield was shot; he responded by telegram from Paris. Sims' recommendations later gained acceptance.
- Gallbladder surgery: In 1878, Sims drained a distended gallbladder and removed its stones. He published the case believing it was the first of its kind; however, a similar case had already been reported in Indianapolis in 1867
|
WINNERS QUIZ NUMBER 11 |
FIRST PRIZE
Dr. Kiran Dabholkar
9167008017 |
SECOND PRIZE
Dr. Zubin Sheriar
zubinsheriar@gmail.com |
THIRD PRIZE
Dr. Prachi Risbud
7738535015 |
FOURTH PRIZE
Dr. Shashikant Avhad
bharat00586@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 10 |
Ovarian cycle can be correlated with all except: |
a. Endometrial sampling |
b. Vaginal cytology |
c. Blood hormonal levels |
d. Estrous cycle |
ANSWER NUMBER 10 |
d. Estrous cycle |
Explanation :
Ovarian cycles consists of the following : recruitment & growth of the follicles, ovulation, corpus luteum formation and finally regression of corpus luteum. Because of the changes in the ovary there are simultaneous changes in the uterus (endometrium)-the menstrual cycle. Endometrial sampling and proliferative or secretory endometrium will tell whether the female has ovulated or not. Similarly the hormones FSH, LH, Estradiol, and progesterone will also correlate with the phases of ovarian cycle. Vaginal cytology & calculating the maturation index will tell us the phase of the ovarian & menstrual cycle. Estrous cycle DOES NOT occur in human beings. |
The estrous cycle comprises of the recurring physiologic changes that are induced by reproductive hormones in most mammalian placental females. Humans undergo a menstrual cycle instead. Estrous cycles start after puberty in sexually mature females and are interrupted by anestrous phases or pregnancies. Typically estrous cycles continue until death. Animals that have estrous cycles reabsorb the endometrium if conception does not occur during that cycle. Animals that have menstrual cycles shed the endometrium through menstruation instead. Another difference is sexual activity. In species with estrous cycles, females are generally only sexually active during the estrus phase of their cycle. This is also referred to as being “in heat.” In contrast, females of species with menstrual cycles can be sexually active at any time in their cycle, even when they are not about to ovulate. |
WINNERS QUIZ NUMBER 10 |
FIRST PRIZE
Dr. Nisha Krishnan
9076547783 • nisha.obgy@gmail.com |
SECOND PRIZE
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com |
THIRD PRIZE
Dr. Dhaval Belvi
9833700719 |
FOURTH PRIZE
Dr. Devika Desai
9561106644 / 9594759110 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 9 |
A newborn with 46XX has external genitalia of male. All of the following are the possible causes except: |
a. Placental aromatase deficiency |
b. Maternal androgen adrenal tumor |
c. Anti-Mullerian hormone (AMH) deficiency |
d. Wnt4 mutation |
ANSWER NUMBER 9 |
c. Anti-Mullerian hormone (AMH) deficiency |
Explanation: The baby has karyotype of 46 XX and external genitalia of male. So this is a case of female pseudohermaphroditism.
Causes of female pseudohermaphroditism are: |
- Congenital adrenal hyperplasia.
- Elevated androgens in the maternal circulation which cross the placenta and cause virilization of the external genitalia.
- Placental aromatase deficiency.
- Wnt4 mutation. Wnt4 Mullerian aplasia and ovarian dysfunction is a disorder that occurs in females and affects the reproductive system. This condition is caused by abnormal development of the Mullerian duct. Individuals with Wnt4 Mullerian aplasia and ovarian dysfunction typically have an underdeveloped or absent uterus and may also have abnormalities of other reproductive organs. Women with this condition have normal breast and public hair development and primary amenorrhea. Women with Wnt4 Mullerian aplasia and ovarian dysfunction have higher than normal levels of androgens in their blood. These high levels of androgens cause acne, hirsutism and virilization. Kidney abnormalities may be present in some affected individuals.
|
AMH DEFICIENCY= PMDS=UTERINE HERNIA SYNDROME
Karyotype = 46 XY and normal male external genitalia.
Persistent Mullerian duct syndrome (PMDS) refers to the presence of a uterus and sometimes other Mullerian duct derivatives in a male. PMDS typically is an autosomal recessive disorder.
Typical features include cryptorchidism and the presence of a small, underdeveloped uterus in a male. This condition is usually caused by deficiency of fetal anti-Mullerian hormone (AMH) effect due to mutations of the gene for AMH or the anti-Mullerian hormone receptor. |
WINNERS QUIZ NUMBER 9 |
FIRST PRIZE
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com |
SECOND PRIZE
Dr. Shilpa Agrawal
drshilpaagrawal@yahoo.co.in |
THIRD PRIZE
Dr. Pravina Gandhi
9820190257 |
FOURTH PRIZE
Dr. Freni Shah
frenishah9488@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 8 |
Precocious puberty associated with bony dysplasia and café au lait spots on skin are seen in: |
a. Laurence-Moon-Biedl syndrome |
b. McCune-Albright syndrome |
c. Alport’s syndrome |
d. Frohlich’s syndrome |
ANSWER NUMBER 8 |
b. McCune-Albright syndrome |
Precocious puberty associated with bony dysplasia and café au lait spots on skin is seen in McCune-Albright syndrome.
It is a GnRH-independent/pseudoprecocious puberty in which ovary is the source of estrogen.
|
Laurence-Moon-Biedl syndrome: hypogonadotropic hypogonadism (hypothalamic amenorrhea), mental retardation, polydactyly, and retinitis pigmentosa Frohlich’s syndrome: hypogonadotropic hypogonadism, obesity, and genital hypoplasia Alport’s syndrome: anterior lenticonus, glomerulonephritis, and hematuria In options a and d, there is delayed/absent puberty.
|
WINNERS QUIZ NUMBER 8 |
FIRST PRIZE
Dr. Misbah Inamdar
9920134423 • miisbah23@gmail.com |
SECONDPRIZE
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com |
THIRD PRIZE
Dr. Kiran Dabholkar
9167008017 |
FOURTH PRIZE
Dr. Dhaval Belvi
9833700719 • drdhavalbelvi@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 7 |
You are delivering an obese primigravida at 41 weeks. After 15 min of pushing, the baby’s head delivers spontaneously but then retracts back against the perineum. As you apply gently downward traction to the head, the baby’s anterior shoulder fails to deliver. All of the following are appropriate next steps in the management of this patient, except: |
a. Instruct the nurse to apply fundal pressure |
b. Cut a generous episiotomy |
c. Flex the maternal legs upon her abdomen |
d. Apply supra pubic pressure |
ANSWER NUMBER 7 |
a. Instruct the nurse to apply fundal pressure |
Explanation:
In this clinical scenario, a shoulder dystocia is encountered. Shoulder dystocia becomes obvious when the fetal head emerges and then retracts against the perineum, commonly referred to as the “turtle sign.” |
A shoulder dystocia occurs when the fetal shoulders fail to spontaneously deliver secondary to impaction of the anterior shoulder against the pubic bone, after delivery of the head has occurred. Shoulder dystocia is an obstetric emergency, and one should always call for help when such a situation is encountered. A generous episiotomy should always be made to allow the obstetrician to have adequate room to perform a number of manipulations to try to relieve the dystocia. Such maneuvers include the following: suprapubic pressure, McRoberts maneuver (flexing maternal legs upon the abdomen and abduct them), Wood’s corkscrew maneuver (rotating the posterior shoulder), and delivery of the posterior shoulder. |
There is no role for fundal pressure because this action further impacts the shoulder against the pubic bone and makes the situation worse. Never give fundal pressure in cases of shoulder dystocia. |
WINNERS QUIZ NUMBER 7 |
FIRST PRIZE
Dr Nandini Ram Babu
9892911137 |
SECOND PRIZE
Dr. Shilpa
9223280008 • drshilpaagrawal@yahoo.co.in |
THIRD PRIZE
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com |
FOURTH PRIZE Dr. Freni Shah
frenishah9488@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 6 |
27 years old obese female complaints of amenorrhea of 6 months following her abortion at 16 weeks of gestation due to cervical incompetence.
Now her UPT is negative. Progesterone challenge test is negative & Estrogen + progesterone test is also negative. Likely diagnosis is |
a. PCOS |
b. Premature ovarian failure |
c. Hypothalamo- Pituitary failure |
d. Asherman's syndrome |
ANSWER NUMBER 6 |
d. Asherman's syndrome |
Explanation:
In patients with secondary amenorrhea, after ruling out pregnancy, progesterone challenge test is to be done.
Patients with anovulation will get menses with progesterone.
If the patient does not get menses with progesterone then E + P challenge test is done.
Patients with pituitary failure and ovarian failure will get menses with E + P.
|
Absence of withdrawal by E + P indicates end organ failure.(Asherman syndrome)
The patient had a second-trimester abortion, following which a curettage may have been done to remove the retained products leading to Asherman syndrome. |
WINNERS QUIZ NUMBER 6 |
FIRST PRIZE
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com |
SECOND PRIZE
Dr. Nandini Ram Babu
9892911137 • atg2016nandini@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 5 |
 |
This CTG is most likely to be seen in which condition ? |
a. fetus is sleeping |
b. ruptured vasa previa |
c. cord compression |
d. Excess of Epidural analgesia |
e. All of the above |
ANSWER NUMBER 5 |
Ruptured vasa previa |
The CTG shows " Sinusoidal Pattern" seen in following cases :
Fetal anemia ( ruptured vasa previa, abruption,fetal hemolysis etc)
Chorioamnionitis
Severe fetal asphyxia
Morphine administration to mother |
WINNERS QUIZ NUMBER 5 |
FIRST PRIZE
Dr. Pooja Kale 9029080799 •
kpoo87@gmail.com |
SECOND PRIZE
Dr. Shilpa Agrawal Jaslok Hospital 9223280008 •
drshilpaagrawal@yahoo.co.in |
Winners Please Contact MOGS Office |
QUIZ NUMBER 4 |
A baby is born with the following birth defects: Craniofacial defects, cleft palate, cardiac defects, hydrocephalus & thymic defects.
Identify the teratogen. |
a) DES (Diethylstilbestrol) |
b) Misoprostol |
c) Isotretinoin |
d) Cocaine abuse |
ANSWER NUMBER 4 |
The correct answer is (c) Isotretinoin. Following are the teratogenic effects : |
Isotretinoin : Craniofacial defects, cleft palate, cardiac defects, hydrocephalus, thymic defects |
DES :Clear cell adenocarcinoma of cervix/vagina, ectropion and adenosis, hypoplastic T-shaped uterus, cervical collars, hoods, septa, withered fallopian tubes; in male fetuses epididymal cysts, microphallus, cryptorchidism, testicular hypoplasia, hypospadias |
Misoprostol : Moebius syndrome |
Cocaine :Placental abruption, abortions, stillbirth, skull defects, cutis aplasia, porencephaly, ileal atresia, cardiac anomalies and visceral infarcts, urinary defects, periventricular leukomalacia, prune-belly syndrome |
WINNERS QUIZ NUMBER 4 |
FIRST PRIZE
Dr. Ranjeet
rajneet1910@gmail.com |
SECOND PRIZE
Dr. Freni Shah
frenishah9488@gmail.com |
Winners Please Contact MOGS Office |
QUIZ NUMBER 3 |
IDENTIFY THIS STALWART
HINT : C.SECTION |
 |
ANSWER NUMBER 3 |
Ferdinand Adolf Kehrer (February 16, 1837 – June 16, 1914) was a German gynaecologist
Kehrer is remembered for performing the first modern Caesarean section. It involved a transverse incision of the lower segment of the uterus
On September 25, 1881, he performed the first modern C-section. The patient was a 26-year-old woman, and the operation proved to be a success.
|
WINNERS QUIZ NUMBER 3 |
FIRST PRIZE
Dr. Kiran Dabholkar
Cell: 9167008017
kmdabholkar@gmail.com |
SECOND PRIZE
Dr Pooja Kale
Cell: 9029080799
kpoo87@gmail.com
|
Winners Please Contact MOGS Office |
QUIZ NUMBER 2 |
A primigravida had developed varicella infection 3 days before her delivery. Which of the following statement is true? |
a. The baby will develop congenital varicella syndrome |
b. There is no risk of infection to the baby |
c. Mother should be given the vaccine and immunoglobulin before delivery |
d. Immunoglobulin should be given to the neonate |
ANSWER NUMBER 2 |
d. Immunoglobulin should be given to the neonate
Explanation :
Administration of varicella-zoster immunoglobulin (VZIG) prevents or attenuates varicella infection in exposed susceptible individuals if given within 96 hours of viral exposure.
The center for Disease Control and Prevention recommends VZIG for immunocompromised susceptible adults who are exposed to varicella, and it should be strongly considered for all susceptible pregnant women (not when the women has already developed the infection).
An attenuated live-virus vaccine (Varivax) was approved for use in 1995.
The vaccine is not recommended for pregnant women.
Maternal varicella during the first half of pregnancy may cause congenital malformations which include chorioretinitis, cerebral cortical atrophy, hydronephrosis, and cutaneous and bony leg defects.
There is no clinical evidence of congenital varicella infection after 20 weeks of gestation.
Fetal exposure to the virus just before or during delivery, and therefore before maternal antibody has been formed, poses a serious threat to newborns.
The incubation period for varicella infection is short, usually less than 2 weeks. In some instances, neonates develop disseminated visceral and central nervous system disease, which is commonly fatal.
For this reason, VZIG should be administered to neonates whenever the onset of maternal disease is within about 5 days before or after delivery. |
WINNERS QUIZ NUMBER 2 |
FIRST PRIZE
Dr. Freni Shah frenishah9488@gmail.com |
SECOND PRIZE
Dr. Siddesh Iyer
Cell: 9223524599 |
Winners Please Contact MOGS Office |
QUIZ NUMBER 1 |
A 16 year old girl was brought with primary amenorrhoea. Her mother mentioned that she started developing breast at the age of 12. She was prescribed OCPs 2 years back by a doctor with no effect. She was having normal stature and was a football player. On examination breast was well developed (Tanner’s stage 5) and pubic hair was minimal (Tanner’s stage 1). What is the diagnosis ? |
ANSWER NUMBER 1 |
Androgen Insensitivity syndrome/Testicular feminization syndrome.
X-linked recessive disorder.
External genitalia looks normal (like female)
Adequate breast development without axillary and pubic hair (or minimal axillary and pubic hair)
Primary amenorrhea and absent uterus ( and hence will not respond to OCPs)
Gonads (testes) are placed in either labia or inguinal canal, or are intra-abdominal
Karyotype XY
Mullerian agenesis patients will also have primary amenorrhea & absent uterus ( & hence will not respond to OCPs) but have normal well developed axillary and pubic hair .
|
WINNERS QUIZ NUMBER 1 |
FIRST PRIZE
Dr Pooja S Kale
Cell: 9029080799 • kpoo87@gmail.com |
SECOND PRIZE
Dr Shital Dilip Potdar
Cell: 09870391024 • potdar.shital85@gmail.com |
Winners Please Contact MOGS Office |
MOGS Quiz Answers and Winners
Dr. Kiran Dabholkar
9167008017
Dr Razia Mansuri
9820739507 • drraziamansuri@rediffmail.com
Dr Sameen Mansuri
8108087304 • sameen2001@rediffmail.com
Dr Madhuri V Joshi
09869046424
Dr. Prachi Risbud
7738535015
Dr. Kiran Dabholkar
9167008017
Dr Shilpa Agrawal
9223280008
Dr. Nandini Ram Babu
9892911137
Dr. Prachi Risbud
7738535015
Dr. Razia Mansuri
9820739507 • drraziamansuri@rediffmail.com
Dr. Sameen Mansuri
8108087304 • sameen2001@rediffmail.com
Dr. Nandini Ram Babu
9892911137
Dr. Isha Jain
9425110522
Dr. Razia Mansuri
9820739507 • drraziamansuri@rediffmail.com
Dr. Sameen Mansuri
8108087304
Dr. Razia Mansuri
9820739507 • drraziamansuri@rediffmail.com
Dr. Sameen Mansuri
8108087304 • sameen2001@rediffmail.com
Dr. Kiran Dabholkar
9167008017
hypoxia” model has been proposed. This model states that the activities that first appear embryologically are the last to
disappear with progressively worsening hypoxia.
with progressively worsening hypoxia. Fetal movements develop after fetal tone (at 9 weeks) and then fetal breathing (at
20–21 weeks), and finally FHR reactivity, at approximately 28 weeks.
lost) in hypoxic states. This gradual hypoxia concept would help explain the increased incidence of abnormal fetal outcome as more biophysical activities decrease in occurrence and eventually disappear. So finally the persistent and true lack of Fetal Tone would be associated with the highest perinatal death rate.
Dr. Prachi Risbud
7738535015
Dr.Isha Jain
9425110522 • drishabharatjain@gmail.com
Dr. Nandini Ram Babu
9892911137 • atg2016nandini@gmail.com
priyanka honavar
priyankahonavar@yahoo.co.in
Priya Shah
dr_pnshah@icloud.com
Dr. Prachi Risbud
7738535015
Dr Nandini Ram Babu
9892911137 • atg2016nandini@gmail.com
Dr. Prachi Risbud
7738535015
Shilpa Agrawal
drshilpaagrawal@yahoo.co.in
Dr. Kiran Dabholkar
kmdabholkar@gmail.com
Dr. Freni Shah
frenishah9488@yahoo.in
Dr. Siddesh Iyer
siddeshiyer@gmail.com
Dr. Kiran Dabholkar
kmdabholkar@gmail.com
Dr. Prachi Risbud
7738535015
Dr. Shweta More
shwetaankushmore@gmail.com
Dr. Shilpa Agrawal
9223280008
Dr. Ashwini Sakhalkar
ashwinisakhalkar@gmail.com
Dr. Rashmi G. Jalvee
983391883
Dr. Divya R
7039060390 • rdtmc06@gmail.com
Dr. Shilpa Agrawal
9223280008
Dr. Nandini Ram Babu
9892911137 • atg2016nandini@gmail.com
Trial of scar is contraindicated in all EXCEPT :
Kiran Dabholkar
9167008017
Dr. Freni Shah
9869562008
Dr. Prachi Risbud
7738535015
A 30-year-old primigravida at 39 weeks has been completely dilated and has been pushing for 3 h. She had taken epidural analgesia. She is exhausted and has a temperature of 37.8°C. The fetal heart rate is 170/min with decreased variability. The patient’s membranes have been ruptured for over 24 h. The patient’s cervix is completely dilated and effaced, and the fetal head is visible at the introitus between pushes. The fetal bones are at the +3 station. What is the most appropriate next step in the management of this patient?
Shilpa Agrawal
9223280008
drshilpaagrawal@yahoo.co.in
Dr. Freni Shah
9869562008
Dr. Nandini Ram Babu
9892911137
As per FIGO staging this is stage :
Pleural effusion with positive cytology is stage IV A
Kiran Dabholkar
kmdabholkar@gmail.com
Dr. Prachi Risbud
7738535015
Bhavini Shah
doc.bhavini@gmail.com
A 28-year-old lady had Copper T inserted 2 years back. Now Cu-T threads are not seen. USG shows Cu T partly in abdominal cavity. Method of removal is:
Dr. Freni Shah
9869562008
Dr. Shilpa Agrawal
9223280008
Dr. Rashmi G. Jalvee
9833918836
Dr. Kiran Dabholkar
098192 17200
Dr. Freni Shah
frenishah9488@yahoo.in
Dr. Prachi Risbud
7738535015
1. Within 72 hours of fertilization = DC, DA
2. Between 4th & 8th day = MC, DA
3. Between 8th & 12th day = MC, MA
4. After 12 days = conjoint/Siamese twins
Twin peak sign indicated 2 fused placenta (seen in dichorionic twins)
Dr. Freni Shah
frenishah9488@yahoo.in
Dr. Shilpa Agrawal
drshilpaagrawal@yahoo.co.in
Dr. Prachi Risbud
7738535015
Explanation:
Human placenta lactogen (hPL) was named so , because of its potent lactogenic & growth hormone like bioactivity, as well as immunochemical resemblance to human growth hormone. It is detected as early as 2nd or 3rd week after fertilization. It is a single , nonglycosylated polypeptide chain with a molecular weight of 22,279 Da. It is derived from a 25,000 Da precursor. Maternal plasma concentrations are linked to placental mass & they rise steadily till 34-36 weeks of gestation. The hPL production rate near term is by far the greatest of any known hormone in humans- approximately 1gm/day. The half-life in maternal plasma is between 10 & 30 minutes The functions of hPL are:
Dr. Prachi Risbud
7738535015
Dr. Monika Nathani
9075013533
Dr. Nandini Ram Babu
9892911137
Explanation:
Incidence of major congenital malformation in children of diabetic mothers is 5–10%, and most common defects are neural defects (such as anencephaly, spina bifida, and encephalocele) cardiac defects (VSD, transposition of great vessels). Caudal regression syndrome/sacral agenesis is a defect most specific to diabetic embryopathy. Hyperglycemia probably increases the development of free oxygen radicals and interferes with arachidonic acid metabolism, which are responsible for embryopathy.
Major Birth Defects in Infants of Diabetic Mothers :
Dr. Shilpa Agarwal
9223280008
Dr. Kiran Dabholkar
9167008017
Dr. Nirmal khare
9818427823
potentially sparing patients from infertility and early mortality.
The exact etiology of dysgerminomas has not been determined, although recent molecular studies have implicated loss of
function with potential tumor suppressor gene TRC8/RNF139 as a possible etiology.
Germ-cell tumors generally occur in the first 2 decades of life. Epithelial tumors occur in perimenopausal and postmenopausal ladies.
Dr. Nisha Krishnan
9076547783
nisha.obgy@gmail.com
Dr. Shilpa Kaushal
7506012801
shilpakaushal55@gmail.com
Dr. Monika Nathani
monika.nathani@gmail.com
36 weeks pregnant lady complains of bleeding PV since 4 hours. Hb 6gm%, BP-90/60, P/A-uterus tonically contracted, FHS absent. BT=7min & CT= 14min. The likely diagnosis as per Page’s classification is :
Grade 0 :Retrospective diagnosis (after delivery)
Grade 1 : External bleeding, uterine tenderness, and no fetal distress
Grade 2 : Fetal distress or IUFD
Grade 3 : Maternal shock, with or without DIC
Dr. Freni Shah
frenishah9488@yahoo.in
Dr. Rakhi Sikarwar
rakhisikarwar4@gmail.com
Dr. Asmita More
8767460154
asmitamore324@gmail.com
Dr. Saumya Niviti
saumyaniviti03@gmail.com
Endogenous and synthetic oxytocin has a half-life of approximately 3.5 minutes. Carbetocin, in comparison, has a much longer half-life ranging from 85–100 minutes. The elimination half-life following intravenous administration is around 40 minutes Carbetocin should not be used to induce or augment labor since it could cause cardiac or respiratory distress to mother or infant
Dr. Saumya Niviti
9967769717
saumyaniviti03@gmail.com
Dr. Rukhsana Mhate
9820645014
Dr. Ashwini Ingale
8087593134
dr.ashwini_ingle@rediffmail.com
Dr. Sumati Saxena
mailmesumatisaxena@rediffmail.com
Dr. Sonam Kataria
9967769717
sonam_kataria777@yahoo.com
Dr. Kiran Dabholkar
kmdabholkar@gmail.com
Dr. Kranti Phadnis
krantiphadnis@gmail.com
Dr. Sumati Saxena
9452495704
saumyaniviti03@gmail.com
Dr. Bhavini Shah
doc.bhavini@gmail.com
Dr. Parikshit Tank
pariktank@gmail.com
Dr. Deepa Talreja
7506987141
Dr. Jatinder Kaur
9892590464
Causes include :
Dr. Shilpa Agrawal
+91 92232 80008
drshilpaagrawal@yahoo.co.in
Dr. Bhavini Shah
doc.bhavini@gmail.com
Dr. Dhaval Belvi
9820273729
Dr. Sachin Dalal
9833032120
Dr. Dhaval Belvi
9833700719
Dr. Kiran Dabholkar
9167008017
Dr. Rajneet Bhatia
rajneet1910@gmail.com
Dr. Ranjit Inamdar
9820273729
Explanation: Listerial infections are more common in very old or young, pregnant or immunocompromised patients. Listeriosis during pregnancy can have the following effects:
Maternal
Fetal
CMV, Rubella and Toxoplasma infections have a teratogenic effect and are unlikely to cause preterm labor and MSAF.
Dr. Pooja Kale
9029080799
kpoo87@gmail.com
Dr. Bhavini Shah
doc.bhavini@gmail.com
Dr. Shilpa Agrawal
drshilpaagrawal@yahoo.co.in
Dr. Freni Shah
frenishah9488@gmail.com
Kranti Phadnis
krantiphadnis@gmail.com
Dhaval Belvi
9833700719
Bhavini Shah
doc.bhavini@gmail.com
Shilpa Agrawal
drshilpaagrawal@yahoo.co.in
Dr. Freni Shah
frenishah9488@gmail.com
Dr. Shilpa Kaushal
7506012801
Dr. Shilpa
9223280008 • drshilpaagrawal@yahoo.co.in
Dr. Jatinder Kaur
drjatinder75@yahoo.co.in
Dr. Kiran Dabholkar
9167008017
Dr. Zubin Sheriar
zubinsheriar@gmail.com
Dr. Prachi Risbud
7738535015
Dr. Shashikant Avhad
bharat00586@gmail.com
Dr. Nisha Krishnan
9076547783 • nisha.obgy@gmail.com
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com
Dr. Dhaval Belvi
9833700719
Dr. Devika Desai
9561106644 / 9594759110
Causes of female pseudohermaphroditism are:
Karyotype = 46 XY and normal male external genitalia.
Persistent Mullerian duct syndrome (PMDS) refers to the presence of a uterus and sometimes other Mullerian duct derivatives in a male. PMDS typically is an autosomal recessive disorder.
Typical features include cryptorchidism and the presence of a small, underdeveloped uterus in a male. This condition is usually caused by deficiency of fetal anti-Mullerian hormone (AMH) effect due to mutations of the gene for AMH or the anti-Mullerian hormone receptor.
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com
Dr. Shilpa Agrawal
drshilpaagrawal@yahoo.co.in
Dr. Pravina Gandhi
9820190257
Dr. Freni Shah
frenishah9488@gmail.com
Dr. Misbah Inamdar
9920134423 • miisbah23@gmail.com
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com
Dr. Kiran Dabholkar
9167008017
Dr. Dhaval Belvi
9833700719 • drdhavalbelvi@gmail.com
Dr Nandini Ram Babu
9892911137
Dr. Shilpa
9223280008 • drshilpaagrawal@yahoo.co.in
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com
Dr. Freni Shah
frenishah9488@gmail.com
Now her UPT is negative. Progesterone challenge test is negative & Estrogen + progesterone test is also negative. Likely diagnosis is
In patients with secondary amenorrhea, after ruling out pregnancy, progesterone challenge test is to be done.
Patients with anovulation will get menses with progesterone.
If the patient does not get menses with progesterone then E + P challenge test is done.
Patients with pituitary failure and ovarian failure will get menses with E + P.
The patient had a second-trimester abortion, following which a curettage may have been done to remove the retained products leading to Asherman syndrome.
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com
Dr. Nandini Ram Babu
9892911137 • atg2016nandini@gmail.com
This CTG is most likely to be seen in which condition ?
Fetal anemia ( ruptured vasa previa, abruption,fetal hemolysis etc)
Chorioamnionitis
Severe fetal asphyxia
Morphine administration to mother
Dr. Pooja Kale
9029080799 • kpoo87@gmail.com
Dr. Shilpa Agrawal
Jaslok Hospital
9223280008 • drshilpaagrawal@yahoo.co.in
Dr. Ranjeet
rajneet1910@gmail.com
Dr. Freni Shah
frenishah9488@gmail.com
HINT : C.SECTION
Ferdinand Adolf Kehrer (February 16, 1837 – June 16, 1914) was a German gynaecologist Kehrer is remembered for performing the first modern Caesarean section. It involved a transverse incision of the lower segment of the uterus On September 25, 1881, he performed the first modern C-section. The patient was a 26-year-old woman, and the operation proved to be a success.
Dr. Kiran Dabholkar
Cell: 9167008017
kmdabholkar@gmail.com
Dr Pooja Kale
Cell: 9029080799
kpoo87@gmail.com
Explanation :
Administration of varicella-zoster immunoglobulin (VZIG) prevents or attenuates varicella infection in exposed susceptible individuals if given within 96 hours of viral exposure.
The center for Disease Control and Prevention recommends VZIG for immunocompromised susceptible adults who are exposed to varicella, and it should be strongly considered for all susceptible pregnant women (not when the women has already developed the infection).
An attenuated live-virus vaccine (Varivax) was approved for use in 1995.
The vaccine is not recommended for pregnant women.
Maternal varicella during the first half of pregnancy may cause congenital malformations which include chorioretinitis, cerebral cortical atrophy, hydronephrosis, and cutaneous and bony leg defects.
There is no clinical evidence of congenital varicella infection after 20 weeks of gestation.
Fetal exposure to the virus just before or during delivery, and therefore before maternal antibody has been formed, poses a serious threat to newborns.
The incubation period for varicella infection is short, usually less than 2 weeks. In some instances, neonates develop disseminated visceral and central nervous system disease, which is commonly fatal.
For this reason, VZIG should be administered to neonates whenever the onset of maternal disease is within about 5 days before or after delivery.
Dr. Freni Shah
frenishah9488@gmail.com
Dr. Siddesh Iyer
Cell: 9223524599
X-linked recessive disorder.
External genitalia looks normal (like female)
Adequate breast development without axillary and pubic hair (or minimal axillary and pubic hair)
Primary amenorrhea and absent uterus ( and hence will not respond to OCPs)
Gonads (testes) are placed in either labia or inguinal canal, or are intra-abdominal
Karyotype XY
Mullerian agenesis patients will also have primary amenorrhea & absent uterus ( & hence will not respond to OCPs) but have normal well developed axillary and pubic hair .
Dr Pooja S Kale
Cell: 9029080799 • kpoo87@gmail.com
Dr Shital Dilip Potdar
Cell: 09870391024 • potdar.shital85@gmail.com
Dr. Punit Bhojani
MEMBER OF MANAGING COUNCIL
Resi: 24013718 • Cell: 9869727515 • drpunit@hotmail.com